Philip D. Campbell

This study investigates the behavioral effects of isoform-specific NRXN1 deletions in zebrafish, showing unique and overlapping deficits in locomotion, sensorimotor behavior, and social behavior.

This study shows that partial loss of DOT1L causes transcriptional, morphological, and behavioral defects in neurons across multiple models.

This study identifies a single base pair substitution in the syt7a gene in zebrafish linked to an acoustic startle threshold phenotype but shows it does not cause the observed behavioral changes.