Erica Korb

The paper performs transcriptomic analysis of cultured primary mouse cortical neurons to identify sex-specific gene expression patterns and neuropeptide regulation during development.

This paper identifies key factors regulating H2BE incorporation, localization, and eviction in neuronal chromatin, revealing chaperones and complexes involved in its dynamics.

This study shows that low-dose mixtures of endocrine disrupting chemicals alter brain transcriptomes and behavior in mice, with sex-specific effects and changes in cell-cell signaling.

The paper introduces spatial NT-seq to map RNA turnover and stability in the mouse brain, revealing spatial heterogeneity and kinetics scaling mechanisms in response to ECS.

This study shows that H2BE is required for activity-dependent gene expression and homeostatic scaling in neurons.

The paper tests nine ASD-linked transcription regulators in neurons, finding shared gene expression disruptions and firing pattern changes.

The paper identifies persistent chromatin loops in hippocampal neurons after fear conditioning that correlate with gene expression changes during memory recall.

This paper introduces a protein editing technology that enables site-specific insertion of non-canonical residues into endogenous proteins in live mammalian cells.

This study shows that partial loss of DOT1L causes transcriptional, morphological, and behavioral defects in neurons across multiple models.

This study identifies H2BE as a widely expressed H2B variant that enhances chromatin accessibility in neurons, promoting synaptic gene expression and long-term memory.

This study identifies a BLAnoci ensemble in the basolateral amygdala that mediates both acute and chronic pain behavior through a novel amygdala-striatal circuit involving the nucleus accumbens shell.

The paper identifies a shared transcriptional signature in ASD and related disorders through analysis of chromatin modifier depletion in neuronal cultures and mouse models.