HERMINE H. M. MAES

The paper estimates the covariance between liability to a disorder and age at onset in censored populations using a mixture distribution model applied to twin data.

This study identifies 52 genetic variants associated with adolescent-onset depression through a large GWAS, revealing age-specific mechanisms and potential drug targets.

This study performs a multi-ancestral GWAS of DSM-defined nicotine dependence, finding genetic correlations with other substance use disorders and health traits, and identifying a strong general addiction liability factor.

This study uses twin-based Mendelian randomisation to find bidirectional causation between smoking and blood DNA methylation at multiple genomic sites.

This study uses BMI GWAS and genomic structural equation modeling to show that genetic influences on BMI remain stable from age 40 to 73 in men and women.

The paper compares genome-wide data from opioid use and dependence cases to identify genetic associations and differences between exposure and dependence.

This study uses Mendelian randomization to find a causal link between major depression and alcohol dependence using large genetic datasets.