Bhagaban Mallik

This study shows that mitochondrial Complex I depletion in Drosophila neuromuscular junctions leads to opposing effects on pre- and postsynaptic function through ROS-dependent mechanisms.

This study shows that dAsap regulates membrane protrusions in Drosophila S2R+ cells through Arf6 inactivation via its ArfGAP domain and re-localization of actin regulators.

This study identifies dAsap as a regulator of NMJ morphology and synaptic calcium homeostasis in Drosophila through Arf6-dependent signaling.

This study identifies Mitochondrial Complex I subunits as regulators of synaptic transmission and growth in Drosophila, with implications for human neurological disorders.