Author

Fanny Jaudon

Italian Institute of TechnologyORCID 0000-0001-7648-0977Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:electronic health records· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:perturbation response prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

Lorenzo A. CingolaniiD· 2 shared Agnes ThalhammeriD· 1 shared Anna LongattiiD· 1 shared Caroline Bonnet· 1 shared Edoardo MorettoiD· 1 shared Fabio BenfenatiiD· 1 shared Fabrizia CescaiD· 1 shared Federico MiozzoiD· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersNeuroscience

Lorenzo A. CingolaniORCID

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3 papers

Recent·Most cited

NeuroscienceeLifePeer-reviewedJan 30, 2023

The tetraspanin TSPAN5 regulates AMPARs exocytosis by interacting with the AP-4 complex

Edoardo Moretto, Anna Longatti, Federico Miozzo…·Maria Passafaro

The paper identifies TSPAN5 as an adaptor protein that promotes AMPA receptor exocytosis by interacting with AP-4 and Stargazin in rat hippocampal neurons.

11 citations

ratexperimental assay

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NeuroscienceJournal of Cell SciencePeer-reviewedJul 19, 2021

A developmental stage- and Kidins220/ARMS-dependent switch in astrocyte responsiveness to brain-derived neurotrophic factor

Fanny Jaudon, Martina Albini, Stefano Ferroni…·Fabrizia Cesca

This study shows that astrocytes switch from a kinase-based to a calcium-based response to BDNF during development, with Kidins220/ARMS modulating both pathways and regulating Kir4.1 expression.

15 citations

mouseproteomics

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NeurosciencebioRxivPreprintJun 10, 2019

Correction of β3 integrin haplo-insufficiency by CRISPRa normalizes cortical network activity

Fanny Jaudon, Agnes Thalhammer, Lorenzo A. Cingolani

This study uses CRISPRa to correct β3 integrin haplo-insufficiency in neurons, rebalancing network activity without causing hyperactivity seen with exogenous gene expression.

7 citations

mouseelectronic health records

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