Catalina Ribas

This study identifies Gαq as a regulator of mitochondria-lysosome contact dynamics, showing its role in mitochondrial fragmentation and quality control through interactions with Fis1 and RabGAPs.

The paper identifies functionally distinct and pharmacologically targetable cancer-associated fibroblast (CAF) programs in head and neck cancer using proteomic and functional assays.

This study identifies GRK2's role in hair follicle homeostasis by showing its deletion leads to cyst formation, stem cell disruption, and hair loss in mice.

This study shows that loss of Gαq in fibroblasts promotes aggressive oral cancer progression by altering ECM remodeling, exosome secretion, and tumor cell migration.

The paper shows that activating RhoC at cell junctions preserves endothelial barrier function during inflammation by enhancing adherens junctions and stabilizing actomyosin.