Nadia Soussi‐Yanicostas

This study shows that DFP intoxication in zebrafish larvae causes neuronal hyperexcitation, apoptosis, and synaptic imbalances, offering a model for studying OP neurotoxicity and antidote development.

This study shows that microglia in zebrafish tauopathy models become more mobile and change morphology, and their ablation increases Tau hyperphosphorylation.

This study investigates neuronal deficits in zebrafish with Scn1Lab deficiency, finding increased excitatory/inhibitory imbalance and neuron apoptosis linked to epileptiform seizures.

The paper shows that reduced Wnt/β-catenin signaling in microglia of the developing brain causes pro-inflammatory activation and white matter injury, and that restoring this pathway can prevent these effects.