Juliette Van Steenwinckel

This study shows that C-sections combined with systemic inflammation worsen brain and gut issues in a preterm mouse model, increasing neurodevelopmental risks.

This study investigates the long-term metabolic effects of perinatal inflammation in mice, finding sex-dependent glucose metabolism dysregulation and potential therapeutic implications.

The paper shows that neuroinflammation in OPCs causes major transcriptomic changes by exploiting a primed epigenetic landscape rather than altering chromatin accessibility, leading to white matter injury.

This study investigates the effects of perinatal inflammation on cerebellar development in a mouse model of encephalopathy of prematurity, finding unique microglia activation and type I interferon signaling dysregulation in the cerebellum.

This study shows that perinatal IL-1-induced inflammation reduces Tbr2+ progenitor cell proliferation in the hippocampus, leading to long-term anxiety and spatial memory deficits in mice.

The paper shows that reduced Wnt/β-catenin signaling in microglia of the developing brain causes pro-inflammatory activation and white matter injury, and that restoring this pathway can prevent these effects.

This paper identifies DLG4 (PSD95) expression in microglia and links genetic variation in DLG4 to white matter structure in preterm infants.