Author

Doriane Pereira

Centre National de la Recherche ScientifiqueORCID 0000-0001-7846-8541Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Physiology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Organism:mouse· 3 Approach:experimental assay· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

Edgar JauliaciD· 3 shared Maxime Di GalloiD· 3 shared Stéphanie Backer· 3 shared Athanassia SotiropoulosiD· 2 shared Florian Alexis BrittoiD· 2 shared Gaspard MacauxiD· 2 shared Matthieu Dos SantosiD· 2 shared Pascal MaireiD· 2 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersPhysiology

Edgar JauliacORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintSep 14, 2026

REDD1 Regulates MERCS, Protein Synthesis and NMJ Stability in Fast Myofibers During Dexamethasone-Induced Muscle Wasting

Doriane Pereira, Frédéric Simon, Maxime Di Gallo…·Florian Alexis Britto

DOI 10.64898/2026.09.07.749905

mousebulk RNA-seq

𝕏 PostBluesky

PhysiologybioRxivPreprintFeb 17, 2026

c-MAF transduces fast motor neuron firing to sustain fast-glycolytic myofibers and neuromuscular junctions

Edgar Jauliac, Stéphanie Backer, Shunya Sadaki…·Pascal Maire

This study identifies c-MAF as a mediator of motoneuron activity in maintaining fast-glycolytic muscle fibers and neuromuscular junctions through chromatin accessibility changes and gene regulation.

2 citations

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PhysiologybioRxivPreprintFeb 5, 2026

SIX1-dependent myofiber typology and metabolism controls muscle hypertrophy

Maxime Di Gallo, Léa Delivry, Doriane Pereira…·Florian Alexis Britto

This study shows that SIX1 regulates muscle fiber hypertrophy by modulating metabolism and fiber type transitions in response to mechanical overload.

1 citation

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