Pascal Maire

The paper introduces CellHub Database, a consolidated ligand-receptor interaction resource for cell-cell communication analysis across multiple species.

This study identifies AR-dependent Mybph repression as a mechanism contributing to skeletal muscle sexual dimorphism using ChIL-seq and an AirID-AR knock-in mouse.

Cell-Hub provides a user-friendly interface for single-cell RNA sequencing analysis, integrating multiple tools to support all essential analysis steps without requiring programming expertise.

This study investigates the role of EYA1/EYA2 and EYA3/EYA4 in skeletal myogenesis, showing that EYA3/EYA4 are critical for adult muscle regeneration while EYA1/EYA2 are essential for embryonic development.

The paper identifies a new population of myonuclei during skeletal muscle hypertrophy using snRNA-seq and RNAscope, showing distinct localization and gene expression profiles.

This study identifies c-MAF as a mediator of motoneuron activity in maintaining fast-glycolytic muscle fibers and neuromuscular junctions through chromatin accessibility changes and gene regulation.

This study shows that SIX1 regulates muscle fiber hypertrophy by modulating metabolism and fiber type transitions in response to mechanical overload.

The paper identifies local translational regulation at the muscle-tendon junction (MTJ) and shows that KLF-family transcription factors are translationally upregulated there, driving domain-specific gene expression.

This paper describes a protocol for analyzing muscle fiber type and cross-sectional area in cryosections of whole lower mouse hindlimbs using immunofluorescence and histochemistry.

This paper identifies DiPRO1 as a regulator of myoblast differentiation and mesenchymal tumor suppression through epigenetic and transcriptional mechanisms, showing its potential as a therapeutic target.

This study identifies LRRFIP2 as a negative regulator of HIF-1 activity in cardiomyocytes, showing its role in modulating hypoxia response during embryonic heart development.

This paper identifies SIX1 as a critical transcription factor downstream of mesenchymal signals that cooperates with RUNX1 and SMAD4 to regulate {Delta}Np63 expression in Mullerian duct epithelium, leading to vaginal stratified squamous epithelium formation and preventing adenosis.