Eva Kaufmann

This paper develops a novel mouse model of cat allergic rhinitis that recapitulates key clinical features and enables mechanistic and therapeutic studies.

This study shows β-glucan reprograms neutrophils to induce disease tolerance against Influenza A virus by promoting a unique subset of neutrophils that limit tissue damage and enhance survival.

This study identifies Cnes2b as a regulator of host resistance and inflammation in response to Cryptococcus deneoformans infection, showing sex-dependent effects on fungal burden and tissue damage.

The paper identifies basophils as a source of oncostatin M that activates vagal pruriceptor neurons during allergic responses.