Author

Annie Beauchamp

Research Institute of the McGill University Health Centre

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:metagenomics· 1

Frequent Co-Authors

Donald Cuong VinhiD· 2 shared Isabelle Angers· 2 shared A. Kevin Watters· 1 shared Alexi Wloski· 1 shared Anna PereziD· 1 shared Benjamin Mak· 1 shared Carolina AriasiD· 1 shared Dao M. NguyeniD· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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2 shared papersImmunology

Donald Cuong VinhORCID

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Author profile →

3 papers

Recent·Most cited

Human RIG-I deficiency confers susceptibility to Kaposi Sarcoma via loss of latency control

Lucie Roussel, Stéphane Bernier, Mélanie J. Langelier, Yichun Sun, Irem Ulku, Benjamin Mak, Yongbiao Li, Anna Perez, Alexi Wloski, Isabelle Angers, Lily-Rose Vinh, Annie Beauchamp, Sarah Boissel, A. Kevin Watters, Simon Rousseau, , Donald Cuong Vinh

This study identifies RIG-I deficiency as a cause of classic Kaposi sarcoma by impairing control of KSHV infection and reactivation.

bioRxiv · Sep 19, 2025 · PreprintImmunology

Evasion of neutrophil-mediated bacterial clearance in Pseudomonas aeruginosa isolates from new-onset infections in cystic fibrosis children

Kelly Kwong, Sophia Goldman, Annie Beauchamp, Julien Karim Malet, Inès Levade, Lucia Grana, David S. Guttman, Valerie J. Waters, Dao M. Nguyen

This study shows that Pseudomonas aeruginosa isolates from CF patients who failed eradication therapy resist neutrophil-mediated clearance due to loss of Type IV pilus-mediated twitching motility, contributing to infection persistence.

bioRxiv · Sep 29, 2024 · PreprintMicrobiology

Cnes2b Regulates Host Resistance, Inflammatory Responses and Tissue Damage Following Cryptococcus deneoformans Infection

Isabelle Angers, Annie Beauchamp, Marwa El Sheikh, Eva Kaufmann, Donald Cuong Vinh, Salman T. Qureshi

This study identifies Cnes2b as a regulator of host resistance and inflammation in response to Cryptococcus deneoformans infection, showing sex-dependent effects on fungal burden and tissue damage.

bioRxiv · Sep 14, 2024 · PreprintImmunology