Author

Ulrich Rüther

Heinrich Heine University DüsseldorfORCID 0000-0001-8184-7446Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Organism:Xenopus· 1

Frequent Co-Authors

Antonia WiegeringiD· 2 shared Christoph GerhardtiD· 2 shared Christiane OttiD· 1 shared Monika Böddeker· 1 shared Paniz Adibi· 1 shared Peter WalentekiD· 1 shared Sandra HägeleiD· 1 shared Sophie SaunieriD· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCell Biology

Antonia WiegeringORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintJan 8, 2026

The primary cilium controls programmed cell death via its proteasome-regulating function

Antonia Wiegering, Sandra Hägele, Stefanie Kuschel…·Christoph Gerhardt

The paper shows that primary cilia regulate programmed cell death by controlling the proteasomal degradation of MOAP1 through ciliopathy proteins like RPGRIP1L, TCTN1, and CEP290.

DOI 10.64898/2026.01.07.698216

mousehuman

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Developmental BiologybioRxivPreprintFeb 10, 2020

Gain-of-function mutation in Gli3 causes ventricular septal defects

Antonia Wiegering, Paniz Adibi, Ulrich Rüther, Christoph Gerhardt

The paper shows that a gain-of-function mutation in Gli3 causes ventricular septal defects by impairing PDGFR signaling, suggesting potential therapeutic targets.

2 citations

mouseexperimental assay

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