David Greenstein

The paper identifies a uORF in the lin-41 5'UTR that modulates Let-7 microRNA regulation of lin-41 translation in C. elegans.

The paper identifies SPN-4 as a specificity factor for CCR4-NOT-dependent clearance of maternal mRNAs in C. elegans embryos, showing its role in targeting specific transcripts for decay.

This study identifies a sperm-oocyte protein complex, SPE-11 and OOPS-1, essential for meiotic completion, polyspermy block, and eggshell formation in C. elegans.

The paper shows that gap-junctional signaling via innexins controls the positioning of distal somatic cells in C. elegans gonads without affecting germline stem cell boundaries.

The paper shows that ETC-1 promotes proteasome processivity to degrade oocyte regulatory proteins during the oocyte-to-embryo transition in C. elegans.

The paper shows malonyl-CoA is transported from the soma to the germline via gap junctions in C. elegans to support embryogenesis.

The paper shows that reducing torsinA homolog OOC-5 function rescues sterility and premature aging in C. elegans laminopathy mutants.

This study investigates the role of DDX41 mutations in myelodysplastic syndromes using C. elegans SACY-1, revealing antimorphic activity and transcriptome changes through splicing-dependent and -independent mechanisms.

The paper investigates how LIN-41 is degraded during meiotic maturation in C. elegans, identifying degradation domains and ubiquitin ligase involvement.