Author

Karen L. Edelblum

Rutgers New Jersey Medical SchoolORCID 0000-0001-8288-4898Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell signaling & trafficking· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:cell type annotation· 1 Assay:imaging· 1

Frequent Co-Authors

Matthew A. FischeriD· 2 shared Alyssa M. Laguerta· 1 shared Ananya Parthasarathy· 1 shared Andrew Fong· 1 shared Charles A. ParkosiD· 1 shared Harsimran KauriD· 1 shared Jia LuoiD· 1 shared Ken S. LauiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersImmunology

Matthew A. FischerORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Epithelial and γδ T cell CD47 have complementary yet distinct roles in regulating γδ intraepithelial lymphocyte migration

Ananya Parthasarathy, Matthew A. Fischer, Charles A. Parkos, Karen L. Edelblum

This study investigates the roles of CD47 on epithelial cells and γδ T cells in regulating γδ IEL migration, finding complementary yet distinct functions.

bioRxiv · Aug 24, 2026 · PreprintImmunology

Microbiota promote enhanced CD39 expression in γδ intraepithelial lymphocytes through the activation of TCR and IL-15 signaling

Sara Alonso, Harsimran Kaur, Jia Luo, Mai-Uyen Nguyen, Alyssa M. Laguerta, Andrew Fong, Neema Skariah, Rafael José Argüello, Michael Paul Verzi, Mahima Swamy, Ken S. Lau, Karen L. Edelblum

This study shows that microbiota enhance CD39 expression in γδ IELs via TCR and IL-15 signaling, leading to regulatory IEL expansion and reduced inflammation.

Mucosal Immunology · Jul 17, 2025Peer-reviewedImmunology

Type I interferon induces TCR-dependent and -independent antimicrobial responses in γδ intraepithelial lymphocytes

Matthew A. Fischer, Luo Jia, Karen L. Edelblum

This study shows type I interferon induces antimicrobial responses in γδ IELs both independently and dependently on TCR signaling.

bioRxiv · Mar 11, 2024 · PreprintImmunology