Rachel Yehuda

The paper identifies stress-interactive eQTLs that dysregulate gene expression in the brain under stress, uncovering novel genes linked to brain disorder risk in traumatic stress contexts.

The paper examines polygenic risk scores for PTSD severity and chronicity in World Trade Center responders, finding significant genetic contributions and associations with brain imaging phenotypes.

The paper develops a computational model to explore how increased glucocorticoid receptor sensitivity in PTSD contributes to inflammation through HPA-axis and immune system interactions.

This study identifies genetic variants associated with self-reported childhood maltreatment and finds significant genetic correlations with depressive symptoms.

The paper identifies SNRNP35 as a trauma type-specific PTSD gene through analysis of genetically regulated gene expression in PTSD cases and controls.

The paper uses a systems biology approach to analyze how glucocorticoid receptor sensitivity and energy deficit contribute to metabolic dysfunction in PTSD.