Author

Francis J. Doyle

Harvard UniversityORCID 0000-0002-3293-9114Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:electronic health records· 1 Approach:experimental assay· 1 Assay:metabolomics· 1

Frequent Co-Authors

Charles R. MarmariD· 2 shared Janine Dyck FloryiD· 2 shared Marti JettiD· 2 shared Pramod Rajaram SomvanshiiD· 2 shared Rachel YehudaiD· 2 shared Synthia H. MelloniD· 2 shared Aarti GautamiD· 1 shared Bernie J. DaigleiD· 1 shared

Activity by Year

2018-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersSystems Biology

Charles R. MarmarORCID

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3 papers

Recent·Most cited

Role of enhanced glucocorticoid receptor sensitivity in inflammation in PTSD: Insights from a computational model for circadian-neuroendocrine-immune interactions

Pramod Rajaram Somvanshi, Synthia H. Mellon, Rachel Yehuda, Janine Dyck Flory, Linda M. Bierer, Iouri Makotkine, Charles R. Marmar, Marti Jett, Francis J. Doyle

The paper develops a computational model to explore how increased glucocorticoid receptor sensitivity in PTSD contributes to inflammation through HPA-axis and immune system interactions.

American Journal of Physiology-Endocrinology and Metabolism · Apr 21, 2020Peer-reviewedSystems Biology

Mechanistic insights on metabolic dysfunction in PTSD: Role of glucocorticoid receptor sensitivity and energy deficit

Pramod Rajaram Somvanshi, Synthia H. Mellon, Janine Dyck Flory, Duna Abu Amara, Owen M. Wolkowitz, Rachel Yehuda, Marti Jett, Charles R. Marmar, Francis J. Doyle, Leroy Hood, Kai Wang, Inyoul Lee, Rasha Hammamieh, Aarti Gautam, Bernie J. Daigle, Ruoting Yang

The paper uses a systems biology approach to analyze how glucocorticoid receptor sensitivity and energy deficit contribute to metabolic dysfunction in PTSD.

bioRxiv · Dec 17, 2018 · PreprintSystems Biology

Entrainment of circadian rhythms depends on firing rates and neuropeptide release of VIP SCN neurons

Cristina N. Mazuski, John H. Abel, Samantha P. Chen, Tracey O. Hermanstyne, Jeff R. Jones, Tatiana Simon, Francis J. Doyle, Erik D. Herzog

This study identifies how VIP SCN neurons use firing rates and neuropeptide release to entrain circadian rhythms in gene expression and behavior.

Neuron · Jul 14, 2018Peer-reviewedNeuroscience