NeurosciencebioRxivPreprint
ABI3 deletion in TgCRND8 mice is associated with reduced amyloid plaque pathology and altered glial response.
3 citations
Author
Jennifer K. Griffin
University of TorontoORCID 0000-0001-8390-7502Unclaimed ORCID profile · follow to get notified of new preprints
Biology Disciplines
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2020-2023 · peak 2/yrCo-Author Collaboration Network
10 top collaborators · 17 mutual co-author linksFocal author (center)Selected co-authorORCID co-authorUnlinked name
Curved chords = mutual co-authorship3 papers
Recent·Most cited
- NeurosciencebioRxivPreprint
Convergent generation of atypical prions in knock-in mouse models of genetic prion disease
Surabhi Mehra, Matthew E.C. Bourkas, Lech Kaczmarczyk…·Joel C. Watts
This study shows that knock-in mice with specific prion protein mutations spontaneously develop prion disease, producing atypical prion strains with similar properties regardless of mutation type.
3 citations - NeuroscienceAlzheimer s & DementiaPeer-reviewed
Reactive or transgenic increase in microglial TYROBP reveals a TREM2-independent TYROBP-APOE link in wild-type and Alzheimer's-related mice
Mickaël Audrain, Jean‐Vianney Haure‐Mirande, Justyna Mleczko…·Michelle E. Ehrlich
This study shows that TYROBP-APOE signaling in microglia can occur without TREM2 and influences AD pathology in mouse models.
65 citations