Author

Daniel K. Wilton

Boston Children's HospitalORCID 0000-0001-8432-3197Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Arnaud Frouin· 2 shared Beth StevensiD· 2 shared Frederick W. GergitsiD· 2 shared Alanna E. Carey· 1 shared Allie K. Muthukumar· 1 shared Anika Y. KimiD· 1 shared Anna Marie BlomiD· 1 shared Anthony Daggett· 1 shared

Activity by Year

2021-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Arnaud Frouin

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2 papers

Recent·Most cited

NeuroscienceBrain Behavior and ImmunityPeer-reviewedMar 27, 2024

CUB and Sushi Multiple Domains 1 (CSMD1) opposes the complement cascade in neural tissues

Matthew L. Baum, Daniel K. Wilton, Rachel G. Fox…·Beth Stevens

The paper investigates the role of CSMD1 in opposing the complement cascade in neural tissues and links its dysfunction to schizophrenia risk.

48 citationstop 2%

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NeurosciencebioRxivPreprintDec 3, 2021

Microglia Mediate Early Corticostriatal Synapse Loss and Cognitive Dysfunction in Huntingtons Disease Through Complement-Dependent Mechanisms

Daniel K. Wilton, Kevin J. Mastro, Molly D. Heller…·Beth Stevens

This study shows that microglia and complement proteins drive early synapse loss and cognitive deficits in Huntington's disease by eliminating corticostriatal synapses, and that targeting this pathway can prevent these effects.

5 citations

humanproteomics

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