Author

Travis Rush

University of Alabama at BirminghamORCID 0000-0002-6718-9747Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Assay:electronic health records· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Erik D. RobersoniD· 2 shared J. Nicholas CochraniD· 2 shared Jonathan R. RothiD· 2 shared Adam R. Aldaher· 1 shared Jacob S Mesina· 1 shared Jeremy J. DayiD· 1 shared Lori L. McMahoniD· 1 shared Mohammad WaqasiD· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Erik D. RobersonORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Alzheimer's disease risk gene BIN1 induces Tau-dependent network hyperexcitability

Yuliya Voskobiynyk, Jonathan R. Roth, J. Nicholas Cochran, Travis Rush, Nancy V. N. Carullo, Jacob S Mesina, Mohammad Waqas, Rachael M. Vollmer, Jeremy J. Day, Lori L. McMahon, Erik D. Roberson

This study shows that increased BIN1 expression in neurons leads to hyperexcitability, which is modulated by Tau, suggesting a role for BIN1 in Alzheimer's disease pathogenesis.

eLife · Jul 13, 2020Peer-reviewedNeuroscience

A peptide inhibitor of Tau-SH3 interactions ameliorates amyloid-β toxicity

Travis Rush, Jonathan R. Roth, Samantha J. Thompson, Adam R. Aldaher, J. Nicholas Cochran, Erik D. Roberson

The paper develops a peptide inhibitor of Tau-SH3 interactions and shows it reduces Aβ oligomer toxicity in neurons.

Neurobiology of Disease · Nov 4, 2019Peer-reviewedNeuroscience