David Scheuring

Deleting 27 CDIP-candidate genes (26 confirmed CDIPs) and two phytotoxin biosynthesis genes in Botrytis cinerea nearly abolishes apple infection but leaves slow leaf lesions, pointing to redundant, unidentified host-killing factors.

The paper identifies Hip1 as a homolog of PEIE1, showing both immune signaling modulation and EDS1-dependent cell death induction in plants.

The paper identifies Gas5A, a glucanosyltransferase from Botrytis, as a cell death inducing protein in plants, showing it can trigger cell death both as a PAMP and intracellularly.

This study shows that salicylic acid induces apoplast alkalinization via TMK1, leading to growth restriction independent of auxin signaling.

This paper shows that salicylic acid restricts cell elongation and alters vacuolar morphology and pH, independent of NPR receptors and auxin signaling.

This study shows that ERAD machinery regulates PILS protein turnover in plants under non-stressed conditions, linking ER protein homeostasis to growth regulation.

The Chlamydomonas psb28 mutant shows severe PSII assembly defects, and complexome profiling identifies TEF5 as a novel PSII assembly factor involved in CP47 assembly.

This study identifies AtVPS13M1 as a lipid transporter in Arabidopsis thaliana that affects phospholipid degradation during phosphate starvation and localizes to mitochondria.

The paper investigates the virulence of Botrytis cinerea by generating multiple knockout mutants, revealing high redundancy in phytotoxic compounds that drive necrotrophic infection.

This paper introduces a CRISPR/Cas system using ribonucleoprotein complexes and telomere vectors for efficient marker-free genome editing in Botrytis cinerea, achieving high editing rates and enabling multiple edits.