Author

Francis W. Price

Price Vision GroupORCID 0000-0001-9298-0250Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2 Genetic & Genomic Medicine· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:GWAS / association mapping· 1 Assay:GWAS summary statistics· 1

Frequent Co-Authors

Marianne O. PriceiD· 2 shared Anastasia GurinovichiD· 1 shared Anisha KasiiD· 1 shared Anthony J. GriswoldiD· 1 shared Arpan G. MazumderiD· 1 shared Colin Eric WilloughbyiD· 1 shared Eric A. Schon· 1 shared Evan MagarychoffiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Marianne O. PriceORCID

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Author profile →

3 papers

Recent·Most cited

Cell BiologybioRxivPreprintOct 9, 2026

Chronic ER Stress Disrupts Mitochondrial-Associated ER Membrane Integrity in Corneal Endothelial Cells.

Stephanie Lee, Stefan Y. Kim, H Orhan Akman…·Varun Kumar

This study examines how chronic ER stress disrupts mitochondrial-associated ER membrane integrity in corneal endothelial cells, showing increased ER-mitochondrial distance and altered MAM protein expression.

DOI 10.64898/2026.01.09.698664

humanexperimental assay

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Genetic & Genomic MedicineOphthalmology SciencePeer-reviewedMay 21, 2026

Pharmacogenomics of steroid-induced ocular hypertension: relationship to high-tension glaucomas and new pathophysiologic insight

Zeyuan Song, Satyabrata Pany, Shengru Guo…·M. Elizabeth Fini

The paper identifies 44 SNPs associated with steroid-induced ocular hypertension using a pharmacogenomic approach.

DOI 10.1016/j.xops.2026.101239

humanGWAS summary statistics

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Cell BiologybioRxivPreprintApr 11, 2025

Proteasomal Dysfunction results in ER stress, Endo MT, oxidative stress, and apoptotic cell death resulting in Fuchs Corneal Endothelial Dystrophy like features in mice

Subashree Murugan, Sachin Anil Ghag, Viviane Souza de Campos…·Rajalekshmy Shyam

This study shows proteasomal dysfunction causes ER stress, endo-mesenchymal transition, oxidative stress, and apoptosis in mice, leading to FECD-like features.

1 citation

mouseproteomics

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