Vyacheslav M. Labunskyy

This paper shows that manipulating intracellular ATP levels in yeast through synthetic ATP import extends cellular lifespan and reveals distinct metabolic pathways involved in aging.

This study shows that manipulating heme levels in yeast extends lifespan, independent of Hap4, and reveals a link between heme homeostasis and aging.

The paper uses ribosome profiling to show how iron deficiency affects protein translation in yeast by altering gene expression and RNA-binding protein activity.

The paper shows that reducing Cth2 activity in yeast increases lifespan by improving mitochondrial function through reduced repression of iron-related genes.