Author

Haya Alrashidi

University College LondonORCID 0000-0001-9338-3036Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Andrey Y. AbramoviD· 1 shared Anna I. WernickiD· 1 shared Craig LeightoniD· 1 shared Daniela MelandriiD· 1 shared Dario R. AlessiiD· 1 shared Dilan S. AthaudaiD· 1 shared Dmitri A. RusakoviD· 1 shared Gurvir S. VirdiiD· 1 shared

Activity by Year

2022-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Andrey Y. AbramovORCID

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1 paper

Recent·Most cited

Protein aggregation and calcium dysregulation are the earliest hallmarks of synucleinopathy in human midbrain dopaminergic neurons

Gurvir S. Virdi, Minee L. Choi, James R. Evans, Zhi Yao, Dilan S. Athauda, Stephanie Strohbuecker, Raja Sekhar Nirujogi, Anna I. Wernick, Noelia Pelegrina‐Hidalgo, Craig Leighton, Rebecca S. Saleeb, Olga Kopach, Haya Alrashidi, Daniela Melandri, Jimena Perez-Lloret, , Sonia Gandhi

This study uses hiPSCs with SNCA mutations to identify early molecular events in synucleinopathy, showing protein aggregation precedes functional impairments in midbrain dopaminergic neurons.

npj Parkinson s Disease · Nov 24, 2022Peer-reviewedNeuroscience