Lixiang Ma

This paper introduces MAIS, a scalable neural interface system that enables adaptive neuromodulation through brain organoid-MEA integration and plasticity-driven dynamic decoding.

This study identifies polyQ assemblies in HD cells as dynamic structures that regulate Golgi function, showing that mutant HTT disrupts their stability and leads to Golgipathy.

The paper identifies a proliferative microglia aggregate in human fetal brain development that may serve as a secondary microglial formation center.

This study shows that reduced BAG3-mediated autophagy leads to tau accumulation, contributing to neurodegeneration in Drosophila models of CAG and GGGGCC repeat expansions.