Author

Emiliano Matos‐Perdomo

Universidad de La LagunaORCID 0000-0001-9783-3591Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:yeast· 2 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Cristina Ramos-Pérez· 2 shared Félix MachíniD· 2 shared Grant W. BrowniD· 2 shared Jessel Ayra‐PlasenciaiD· 2 shared Michael LisbyiD· 2 shared Marcos Zamora-Dorta· 1 shared Oliver QuevedoiD· 1 shared Sara Medina‐SuáreziD· 1 shared

Activity by Year

2017-2021 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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2 shared papersMolecular Biology

Cristina Ramos-Pérez

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2 papers

Recent·Most cited

Topoisomerase II deficiency leads to a postreplicative structural shift in all Saccharomyces cerevisiae chromosomes.

Jessel Ayra‐Plasencia, Cristina Ramos-Pérez, Silvia Santana‐Sosa, Oliver Quevedo, Sara Medina‐Suárez, Emiliano Matos‐Perdomo, Marcos Zamora-Dorta, Grant W. Brown, Michael Lisby, Félix Machín

This study shows that Top2 deficiency in yeast causes a structural shift in chromosomes after replication, leading to segregation issues without triggering G2/M checkpoints.

Scientific Reports · Jul 22, 2021Peer-reviewedMolecular Biology

Genome-scale genetic interactions position the Mitotic Exit Network as a major antagonist of transient Topoisomerase II deficiency.

Cristina Ramos-Pérez, Jessel Ayra‐Plasencia, Emiliano Matos‐Perdomo, Michael Lisby, Grant W. Brown, Félix Machín

The paper identifies the Mitotic Exit Network as a protective factor against Top2 deficiency by comparing yeast strains with different Top2 alleles.

G3 Genes Genomes Genetics · Aug 24, 2017Peer-reviewedGenetics