Author

Ben Fogelgren

University of Hawaiʻi at MānoaORCID 0000-0001-9882-7839Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Amanda J. Lee· 2 shared Geetika Y. PatwardhaniD· 2 shared Michael A. OrtegaiD· 2 shared B. FujimotoiD· 1 shared Brian E. Hew· 1 shared Chantell Balaan· 1 shared Hanna Kumasaka· 1 shared Jesse B. OwensiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Amanda J. Lee

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintApr 17, 2026

The exocyst is an insulin-sensitive regulator of amyloid precursor protein trafficking and amyloid-beta generation in neurons

Chantell Balaan, Geetika Y. Patwardhan, Rachel K Sachs…·Ben Fogelgren

The paper shows the exocyst regulates APP trafficking and amyloid-beta generation in neurons, with insulin signaling shifting exocyst activity away from APP to GLUT4.

DOI 10.64898/2026.04.14.717551

humanmouse

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Developmental BiologyDisease Models & MechanismsPeer-reviewedAug 25, 2022

Exocyst Inactivation in Urothelial Cells Disrupts Autophagy and Activates non-canonical NF-kB

Michael A. Ortega, Ross Villiger, Malia H. Harrison-Chau…·Ben Fogelgren

This study shows that Exoc5 ablation disrupts autophagy in urothelial cells, activates non-canonical NF-κB signaling, and leads to ureter obstruction.

6 citations

mouseexperimental assay

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