Author

Geetika Y. Patwardhan

University of Hawaiʻi at MānoaORCID 0000-0002-8180-7329Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Amanda J. Lee· 2 shared Ben FogelgreniD· 2 shared Michael A. OrtegaiD· 2 shared B. FujimotoiD· 1 shared Brian E. Hew· 1 shared Chantell Balaan· 1 shared Hanna Kumasaka· 1 shared Jesse B. OwensiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Amanda J. Lee

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2 papers

Recent·Most cited

The exocyst is an insulin-sensitive regulator of amyloid precursor protein trafficking and amyloid-beta generation in neurons

Chantell Balaan, Geetika Y. Patwardhan, Rachel K Sachs, Hanna Kumasaka, Swasthita Sadagopan, Shion Aou, Amanda J. Lee, Luke T. Nelson, Brian E. Hew, Jesse B. Owens, Noémi Polgár, Michael A. Ortega, Robert A. Nichols, Ben Fogelgren

The paper shows the exocyst regulates APP trafficking and amyloid-beta generation in neurons, with insulin signaling shifting exocyst activity away from APP to GLUT4.

bioRxiv · Apr 17, 2026 · PreprintNeuroscience

Exocyst Inactivation in Urothelial Cells Disrupts Autophagy and Activates non-canonical NF-kB

Michael A. Ortega, Ross Villiger, Malia H. Harrison-Chau, Suzanna Lieu, Kadee‐Kalia Tamashiro, Amanda J. Lee, B. Fujimoto, Geetika Y. Patwardhan, Joshua K. Kepler, Ben Fogelgren

This study shows that Exoc5 ablation disrupts autophagy in urothelial cells, activates non-canonical NF-κB signaling, and leads to ureter obstruction.

Disease Models & Mechanisms · Aug 25, 2022Peer-reviewedDevelopmental Biology