Torbjørn Elvsåshagen

This study compares thalamic nucleus volumes across multiple psychiatric and neurological disorders using MRI, finding significant volume reductions in specific regions associated with different conditions.

This study develops a spiking network model of auditory novelty detection and shows how SCZ-related cellular changes impair mismatch negativity.

This study analyzes GWAS data from nearly 1 million cases to show widespread genetic overlap between neurological and psychiatric disorders, with shared biological processes and divergent effect sizes.

The paper uses computational modeling to show schizophrenia-associated changes in ion-channel expression reduce delta-band power in neural simulations.

This study identifies 72 genetic loci associated with basal ganglia volumes and finds significant genetic overlaps with Parkinson's disease, schizophrenia, and migraine.

This study shows that 32 hours of sleep deprivation increases the T1w/T2w ratio in specific cortical regions compared to a normal sleep-wake cycle.

This study identifies 42 genetic loci associated with thalamus and its nuclei volumes and finds genetic overlaps with cortical regions and ten brain disorders.

Meta-analysis of 41,917 European-ancestry bipolar disorder cases versus 371,549 controls yields 64 risk loci (33 novel), neuronal and synaptic gene enrichment, and 15 eQTL-prioritized genes.

The paper assesses LTP-like visual cortical plasticity in schizophrenia and bipolar disorder using VEP modulation, finding impaired N1b modulation in SZ and BDI but not BDII, linked to symptom severity.

This study uses mixed models and advanced diffusion imaging to show that sleep deprivation and normal sleep-wake cycles differentially affect white matter microstructure.

The paper examines VEP modulation in 415 healthy individuals, finding significant effects for several components and age-related correlations, supporting VEP as a measure of synaptic plasticity.

The paper identifies genetic loci associated with brainstem volumes and finds genetic overlaps with psychiatric and neurological disorders.

This paper examines brain aging in major depressive disorder (MDD) using a large dataset, finding that MDD patients show increased brain age differences compared to controls.

The paper examines common genetic variants' contribution to bipolar disorder risk in multiplex families and finds higher polygenic risk scores in affected individuals compared to controls and unaffected family members.

This study identifies 73 genome-wide significant loci for mood disorders, revealing shared and distinct genetic factors between major depressive disorder and bipolar disorder.

The paper estimates brain age gaps using MRI and machine learning in individuals with and without brain disorders, finding heritability and genetic overlap with common disorders.

This study identifies 30 genome-wide significant loci associated with bipolar disorder, including novel genes involved in ion channels, neurotransmitters, and synaptic components.