Deepak Reyon

This study shows that disrupting the kringle 1 domain of prothrombin in zebrafish causes late-onset mortality due to impaired clot formation, revealing its role in thrombin biosynthesis and activation.

This study shows that zebrafish dscaml1 is crucial for retinal patterning and oculomotor circuit function, with mutations causing deficits in gaze stabilization and saccade fatigue similar to human ocular motor apraxia.

The paper critiques the experimental design and data interpretation in a previous study claiming CRISPR-Cas9 causes unexpected mutations, arguing the conclusions are not supported by the evidence.