Author

Manushri Dalvi

George Washington UniversityORCID 0000-0002-0149-7990Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Manushri Dalvi1 paper · Emory University

Biology Disciplines

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Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Aravind MeyyappaniD· 2 shared David MendelowitziD· 2 shared John R. BetheaiD· 2 shared Kelly TammeniD· 2 shared Matthew W. KayiD· 2 shared Melanie EschborniD· 2 shared Pranav PrasooniD· 2 shared Sreejita ArnabiD· 2 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersPhysiology

Aravind MeyyappanORCID

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2 papers

Recent·Most cited

TNFR2 Agonism as a Sex-Specific Therapy for Novel Osteoarthritis-Induced Cardiac Dysfunction

Pranav Prasoon, Kelly Tammen, Rebekah Russo, Aravind Meyyappan, Manushri Dalvi, Román Fischer, Melanie Eschborn, Sreejita Arnab, Luke Brabbee, Lauren Schneider, Kayla L. Nguyen, David Mendelowitz, Matthew W. Kay, John R. Bethea

This study shows that TNFR2 agonism can prevent and reverse OA-induced cardiac dysfunction in female mice and delay systolic decline in males, revealing sex-specific cardiac effects of OA.

bioRxiv · Jul 23, 2026 · PreprintPhysiology

Sex-Specific Characterization of a Novel Osteoarthritis-Induced Heart Failure Model in Mice

Pranav Prasoon, Kelly Tammen, Aravind Meyyappan, Manushri Dalvi, Sreejita Arnab, Melanie Eschborn, Roman Fischer, Matthew W. Kay, David Mendelowitz, John R. Bethea

This study characterizes a novel mouse model of OA-induced heart failure, revealing sex-specific cardiac remodeling and molecular pathways associated with different HF phenotypes.

bioRxiv · Oct 16, 2025 · PreprintMolecular Biology