Author

Saravanan Subramaniam

Boston UniversityORCID 0000-0002-0194-5044Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1 Organism:zebrafish· 1

Frequent Co-Authors

Craig Fletcher· 1 shared Devin KenneyiD· 1 shared Eric BurksiD· 1 shared Florian DouamiD· 1 shared Grace Zhao· 1 shared Hartmut Weiler-GuettleriD· 1 shared Hengky Kurniawan· 1 shared Howard CabraliD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperDevelopmental Biology

Craig Fletcher

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2 papers

Recent·Most cited

Tryptophan metabolism reprogramming contributes to the prothrombotic milieu in mice and humans infected with SARS-CoV-2

Saravanan Subramaniam, Marc Arthur Napoleon, Saran Lotfollahzadeh, Mohamed Hassan Kamel, Hengky Kurniawan, Murad Elsadawi, Devin Kenney, Florian Douam, Markus Bosmann, Stephen A. Whelan, Howard Cabral, Eric Burks, Grace Zhao, Vijaya B. Kolachalama, Katya Ravid, Vipul Chimanlal Chitalia

This study identifies the Kyn-AHR-TF axis as a prothrombotic pathway in SARS-CoV-2 infection, showing increased kynurenine and AHR activation in mice and humans.

bioRxiv · Jan 17, 2025 · PreprintSystems Biology

Coagulation Factor IIIa (f3a) Knockdown in Zebrafish Leads to Defective Angiogenesis and Mild Bleeding Phenotype

Saravanan Subramaniam, Jiandong Liu, Craig Fletcher, Ramani Ramchandran, Hartmut Weiler-Guettler

This paper investigates the role of f3a in zebrafish development, finding that its knockdown causes defective angiogenesis and mild bleeding.

Frontiers in Cell and Developmental Biology · Mar 21, 2022Peer-reviewedDevelopmental Biology