Author

Sarah Phoebeluc Colaiuta

Weizmann Institute of ScienceORCID 0000-0002-9071-9857Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:metabolomics· 1

Frequent Co-Authors

Alexander S. BrandisiD· 2 shared Daniel Kitsberg· 2 shared Giulia CastellaniiD· 2 shared Hannah Partney· 2 shared Javier María Peralta RamosiD· 2 shared Liora Cahalon· 2 shared Michal SchwartziD· 2 shared Miguel Angel AbellanasiD· 2 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Alexander S. BrandisORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Disruption of the Brain-Spleen Axis Impairs Monocyte-Microglia Communication and Accelerates Disease Progression in a Model of Amyloidosis

Tommaso Croese, Miguel Angel Abellanas, Hodaya Polonsky, Michal Arad, Javier María Peralta Ramos, Yuliya Androsova, Serena Riccitelli, Sedi Medina, Francesca Palmas, Romano Strobelt, Giulia Castellani, Denise Kviatcovsky, Sarah Phoebeluc Colaiuta, Miriam Adam, Sama Murad, , Michal Schwartz

This study shows that disrupting the brain-spleen axis accelerates disease progression in amyloidosis by impairing monocyte-microglia communication.

Nature Communications · Jun 19, 2026Peer-reviewedNeuroscience

Alzheimer's disease causes bone marrow myelopoiesis dysfunction

Miguel Angel Abellanas, Leyre Basurco, Maitreyee Purnapatre, Chiara Burgaletto, Giulia Castellani, Sarah Phoebeluc Colaiuta, Javier María Peralta Ramos, Angham Ibraheem, Sama Murad, Paola Antonello, Mariangeles Kovacs, Yuliya Androsova, Bar Nathansohn, Hannah Partney, Liora Cahalon, , Michal Schwartz

This study identifies impaired bone marrow myelopoiesis in Alzheimer's disease, driven by type I interferon signaling, and shows that targeting this pathway improves myeloid function and cognitive outcomes.

bioRxiv · Oct 6, 2025 · PreprintNeuroscience

Accelerated cognitive decline in obese mouse model of Alzheimer's disease is linked to sialic acid-driven immune deregulation

Stefano Suzzi, Tommaso Croese, Adi Ravid, Or Gold, Abbe R. Clark, Sedi Medina, Daniel Kitsberg, Miriam Adam, Katherine A. Vernon, Eva Kohnert, Inbar Shapira, Sergey Malitsky, Maxim Itkin, Alexander S. Brandis, Tevie Mehlman, , Michal Schwartz

This study shows that obesity accelerates cognitive decline in Alzheimer's mice via sialic acid-driven immune changes.

Nature Communications · Mar 9, 2023Peer-reviewedNeuroscience