Marı́a Cecilia Angulo

This study shows that early postnatal interneuron-oligodendroglia communication is essential for PV interneuron myelination, low-gamma oscillations, and fear extinction learning in mice.

This study identifies mitochondrial calcium flux as a critical regulator of oligodendrocyte regeneration, showing its role in proliferation, differentiation, and lesion site repopulation.

The paper introduces an automated workflow and software tools for analyzing oligodendroglial calcium signals in mouse myelin repair models, revealing uncorrelated spontaneous Ca2+ activity in demyelinated lesions.

The paper shows MS-derived oligodendroglia can myelinate axons and form functional connections in a mouse model, suggesting the differentiation block is due to environmental factors, not intrinsic deficits.