Shirley Weiss

This study identifies a neuromodulatory mechanism in Drosophila that enhances stimulus-specific olfactory learning by suppressing unwanted neural responses through axo-axonic connections mediated by mAChR-B.

The paper shows disruption of Ca2+ oscillations in Drosophila perineurial glia leads to seizure-like behavior without affecting BBB structure.

This study shows that glial Ca2+ signaling regulates neuronal excitability through K+ buffering via calcineurin-dependent endocytosis and K2P channel expression in Drosophila.

The paper examines the molecular determinants of synaptic release probability heterogeneity using optical quantal imaging in Drosophila neuromuscular junctions.