Author

Masayoshi Mishina

Ritsumeikan UniversityORCID 0000-0002-1065-9429Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Kenji SakimuraiD· 2 shared Misato YasumuraiD· 2 shared Takeshi UemuraiD· 2 shared Ayako Sugimoto· 1 shared Chiaki Hasegawa· 1 shared Eiko Kato· 1 shared Emi Suzuki‐Kouyama· 1 shared Hiroshi OhiraiD· 1 shared

Activity by Year

2018-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersNeuroscience

Kenji SakimuraORCID

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3 papers

Recent·Most cited

Differential Effects of Inducible Cerebellar Granule Cell and Purkinje Cell Ablation on Motor Coordination and Motor Learning in Adult Mice

Eiko Kato, Misato Yasumura, Shigeyoshi Itohara, Kenji Sakimura, Takeshi Uemura, Masayoshi Mishina

This study compares the effects of selective cerebellar granule cell and Purkinje cell ablation on motor coordination and learning in adult mice, finding that GCs are more critical for motor learning.

bioRxiv · Sep 5, 2025 · PreprintNeuroscience

Neurexins play a crucial role in cerebellar granule cell survival by organizing autocrine machinery for neurotrophins

Takeshi Uemura, Emi Suzuki‐Kouyama, Shiori Kawase, Taiga Kurihara, Misato Yasumura, Tomoyuki Yoshida, Shuya Fukai, Maya Yamazaki, Peng Fei, Manabu Abe, Masahiko Watanabe, Kenji Sakimura, Masayoshi Mishina, Katsuhiko Tabuchi

This study shows that Neurexins are essential for cerebellar granule cell survival by organizing autocrine neurotrophin secretion machinery, independent of postsynaptic contact.

Cell Reports · Apr 1, 2022Peer-reviewedNeuroscience

A transgenic mutant mouse line accompanied by the complete deletion of interleukin-33 showed insulin and leptin resistances

Taku Watanabe, Tomonori Takeuchi, Naoto Kubota, Tasuku Wainai, Keisuke Kataoka, Toshitaka Nakaya, Ayako Sugimoto, Takahiro Sato, Hiroshi Ohira, Ichizo Tsujino, Katsuyoshi Kumagai, Tetsuya Kubota, Chiaki Hasegawa, Kumpei Tokuyama, Kohjiro Ueki, , Takashi Kadowaki

The paper shows that deletion of the Il33 gene in mice leads to insulin and leptin resistance, with improved insulin sensitivity upon IL-33 supplementation.

bioRxiv · Sep 13, 2018 · PreprintMolecular Biology