Author

Maya Yamazaki

Brain Research Institute, Niigata UniversityORCID 0000-0002-3102-9071Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2 Biochemistry· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Kenji SakimuraiD· 2 shared Adam R. RensloiD· 1 shared Adam S. FrostiD· 1 shared Aniliese DealiD· 1 shared Chiayu Q. ChiuiD· 1 shared Christopher P. ArthuriD· 1 shared D. John LeeiD· 1 shared Emi Suzuki‐Kouyama· 1 shared

Activity by Year

2018-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersNeuroscience

Kenji SakimuraORCID

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3 papers

Recent·Most cited

Stereoselective methyl-swapping demonstrates target specificity of cognitive enhancer

Morgane Boone, Udit Dalwadi, Aniliese Deal, Ping Zhu, Tristan Ian Croll, Maya Yamazaki, Kimberly Prescott, Renato Minopoli, Isabella Biscocho, Junhua Wang, D. John Lee, Christopher P. Arthur, Thomas G. Laughlin, Hongyi Zhou, Matthew T. Klope, , Peter Walter

The paper demonstrates that a modified ISRIB analog selectively targets a mutant eIF2B, improving synaptic plasticity and memory in mice, confirming its on-target mechanism.

bioRxiv · Feb 7, 2026 · PreprintBiochemistry

Neurexins play a crucial role in cerebellar granule cell survival by organizing autocrine machinery for neurotrophins

Takeshi Uemura, Emi Suzuki‐Kouyama, Shiori Kawase, Taiga Kurihara, Misato Yasumura, Tomoyuki Yoshida, Shuya Fukai, Maya Yamazaki, Peng Fei, Manabu Abe, Masahiko Watanabe, Kenji Sakimura, Masayoshi Mishina, Katsuhiko Tabuchi

This study shows that Neurexins are essential for cerebellar granule cell survival by organizing autocrine neurotrophin secretion machinery, independent of postsynaptic contact.

Cell Reports · Apr 1, 2022Peer-reviewedNeuroscience

Input-specific NMDAR-dependent potentiation of dendritic GABAergic inhibition

Chiayu Q. Chiu, James S Martenson, Maya Yamazaki, Rie Natsume, Kenji Sakimura, Susumu Tomita, Steven J. Tavalin, Michael J. Higley

The paper shows that NMDAR activation leads to input-specific potentiation of dendritic GABAergic inhibition via somatostatin interneurons, requiring CaMKII and GABA-A β2-subunit, and highlights its role in maintaining synaptic balance.

Neuron · Jan 1, 2018Peer-reviewedNeuroscience