Author

Paola Fabrizio

École Normale Supérieure de LyonORCID 0000-0002-1144-7727Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Organism:C. elegans· 3 Approach:experimental assay· 3 Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1 Assay:metabolomics· 1

Frequent Co-Authors

Francesca PalladinoiD· 3 shared Annie AdraitiD· 1 shared Anupam BhoiiD· 1 shared Cécile BedetiD· 1 shared Grégoire MichauxiD· 1 shared Loïc GelyiD· 1 shared María Jesús Rodríguez‐PaleroiD· 1 shared Marina GiovannettiiD· 1 shared

Activity by Year

2021-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersGenetics

Francesca PalladinoORCID

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3 papers

Recent·Most cited

SIN-3 transcriptional coregulator maintains mitochondrial homeostasis and polyamine flux

Marina Giovannetti, María Jesús Rodríguez‐Palero, Paola Fabrizio, Ophélie Nicolle, Cécile Bedet, Grégoire Michaux, Michael A. Witting, Marta Artal‐Sanz, Francesca Palladino

This study shows that loss of SIN-3 in C. elegans disrupts mitochondrial function and metabolism, leading to mitochondrial fragmentation and altered metabolic flux.

iScience · Apr 22, 2024Peer-reviewedDevelopmental Biology

Loss of SET1/COMPASS methyltransferase activity reduces lifespan and fertility in Caenorhabditis elegans

Matthieu J. Caron, Loïc Gely, Steve Garvis, Annie Adrait, Yohann Couté, Francesca Palladino, Paola Fabrizio

This study shows that reduced H3K4 methylation in C. elegans shortens lifespan and reduces fertility, contradicting previous findings that suggested H3K4 methylation extends lifespan.

Life Science Alliance · Dec 10, 2021Peer-reviewedGenetics

Auxin confers protection against ER stress in Caenorhabditis elegans

Anupam Bhoi, Francesca Palladino, Paola Fabrizio

The paper shows that auxin treatment in C. elegans activates the UPR to confer resistance to ER stress, highlighting potential confounding effects of auxin in studies using the AID system.

Biology Open · Jan 25, 2021Peer-reviewedGenetics