Author

Travis Drow

Seattle Children's Research InstituteORCID 0000-0002-1302-3872Unclaimed ORCID profile · follow to get notified of new preprints

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Travis Drow1 paper · Seattle Children's Research Institute

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Assay:bulk RNA-seq· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Aaron B.I. RoseniD· 1 shared Dane A. Grosvenor· 1 shared David J. RawlingsiD· 1 shared Denny LiggittiD· 1 shared Eric J. AllenspachiD· 1 shared Jacob A. Medjo· 1 shared Jane Hoyt BuckneriD· 1 shared Kimberly A. Dill‐McFarlandiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

Aaron B.I. RosenORCID

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1 paper

Recent·Most cited

Reduced function of the adaptor SH2B3 promotes T1D via altered γc cytokine-regulated, T cell intrinsic immune tolerance

Taylor K. Watson, Aaron B.I. Rosen, Travis Drow, Jacob A. Medjo, Matthew A. MacQuivey, Yan Ge, Denny Liggitt, Dane A. Grosvenor, Kimberly A. Dill‐McFarland, Matthew C. Altman, PATRICK J. CONCANNON, Jane Hoyt Buckner, David J. Rawlings, Eric J. Allenspach

This study identifies SH2B3 as a critical regulator of T cell tolerance in T1D, showing that its deficiency enhances cytokine signaling and promotes islet destruction.

Diabetes · Mar 6, 2025Peer-reviewedImmunology