Hammed A. Badmos

This study identifies macrophage-derived PDGF-BB and GDF-15 as mediators of drug resistance in KRAS-mutant colorectal cancer organoids and shows that targeting tyrosine kinase signaling can restore drug sensitivity.

This study uses Drosophila models to explore how specific genetic mutations in Nigerian colorectal cancer patients affect drug responses, showing that treatment efficacy depends on tumor genotype.