Author

Nemat Khan

Khalifa University of Science and TechnologyORCID 0000-0002-1819-3459Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Trent M. WoodruffiD· 2 shared Avril A. B. RobertsoniD· 1 shared Barry Matthew BradfordiD· 1 shared Clare PridansiD· 1 shared David Arthur HumeiD· 1 shared Dylan Carter‐CusackiD· 1 shared Eduardo A. AlbornoziD· 1 shared Isis Taylor· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersNeuroscience

Trent M. WoodruffORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintJul 23, 2025

Paraquat Licences α-Synuclein to Trigger NLRP3-Dependent Pyroptosis in Microglia

Yuzhihan Cao, Vandana Deora, Nemat Khan…·Eduardo A. Albornoz

This study shows paraquat enables α-synuclein to trigger NLRP3-dependent pyroptosis in microglia, linking environmental toxins to Parkinson's disease pathogenesis.

DOI 10.1101/2025.07.23.666340

mousehuman

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NeuroscienceNeurobiology of DiseasePeer-reviewedNov 22, 2024

The effect of a dominant kinase-dead Csf1r mutation associated with adult-onset leukoencephalopathy on brain development and neuropathology.

Jennifer Stables, Reiss Pal, Barry Matthew Bradford…·David Arthur Hume

This study examines the impact of a heterozygous Csf1r mutation on brain development and neuropathology in mice, showing reduced microglial abundance and altered gene expression in disease models.

9 citations

mousestatistical model

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