Author

Kamil Grycz

Akademia Wychowania Fizycznego im. Eugeniusza Piaseckiego w PoznaniuORCID 0000-0002-1821-2453Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Marcin BączykiD· 3 shared Bartosz WasickiiD· 2 shared Daniel ZytnickiiD· 2 shared Francesco RoselliiD· 2 shared Guillaume CaroniD· 2 shared Simon M. DanneriD· 2 shared Aboud Tahanis· 1 shared Albert Christian LudolphiD· 1 shared

Activity by Year

2024-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersNeuroscience

Marcin BączykORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

NeurosciencebioRxivPreprintSep 4, 2026

Defective synapto-nuclear signaling contributes to motoneuron vulnerability in SOD1-ALS

Kamil Grycz, Cedric Jan, Piotr Zawistowski…·Marcin Bączyk

The paper investigates synapto-nuclear signaling defects in mSOD1 mice and shows that restoring cAMP/PKA signaling mitigates pathology in motoneurons.

DOI 10.64898/2026.09.01.748568

mouseexperimental assay

𝕏 PostBluesky

NeuroscienceProgress in NeurobiologyPeer-reviewedMar 10, 2026

Spinal motoneuron excitability is homeostatically-regulated through β-adrenergic neuromodulation in wild-type and presymptomatic SOD1 mice

Stefano Antonucci, Guillaume Caron, Natalie Dikwella…·Francesco Roselli

This study shows that β-adrenergic receptors regulate spinal motoneuron excitability through homeostatic feedback in both wild-type and presymptomatic SOD1 mice.

5 citationstop 3%

mouseelectronic health records

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 2, 2024

Anodal tsDCS restores the structure and function of the disrupted proprioceptive Ia synapses on spinal motoneurons in the SOD1 G93A mouse model of ALS

T. Jankowiak, Marcin Cholewiński, Katarzyna Kryściak…·Marcin Bączyk

The paper investigates the effects of anodal and cathodal tsDCS on spinal motoneurons in SOD1 G93A mice, finding polarity-dependent changes in synaptic properties but no disease burden reduction.

1 citation

mouseexperimental assay

𝕏 PostBluesky