Author

Laura P. Stabile

University of PittsburghORCID 0000-0002-1822-2707Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Laura P. Stabile1 paper

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Aatur Dilip SinghiiD· 1 shared Adam C. SoloffiD· 1 shared Aditi KulkarniiD· 1 shared Adrian V. LeeiD· 1 shared Alexander Chih-Chieh ChangiD· 1 shared Armando P. SignoreiD· 1 shared Brian Robert IsettiD· 1 shared Carl Kim· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

Aatur Dilip SinghiORCID

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2 papers

Recent·Most cited

Novel transplantable mouse cell line model recapitulates invasive lobular breast carcinoma (ILC) phenotype and immune microenvironment.

Sayali Onkar, Daisong Liu, Darcie D. Seachrist, Jian Zou, Christopher Merkel, Insa Thale, Alexander Chih-Chieh Chang, Linda R. Klei, Jian Chen, Kristen L. Weber-Bonk, Kai Ding, Laura Savariau, Megan E. Yates, Jagmohan Hooda, Laura P. Stabile, , Steffi Oesterreich

The paper develops a novel mouse cell line model of invasive lobular breast carcinoma that recapitulates human tumor phenotype and immune microenvironment, offering a preclinical platform for therapy evaluation.

bioRxiv · Jul 31, 2026 · PreprintCancer Biology

Integrative multi-omic analysis identifies tumor-intrinsic p38 as a driver of immune exclusion in human epithelial cancers

Rebekah Dadey, Krishna Beer Singh, Rose Doerfler, Riley Santiago, Brian Robert Isett, Christopher Deitrick, Carl Kim, Sarah Newman, Marion Joy, Katelyn Smith, Carly Reeder, Armando P. Signore, Ernest Michael Meyer, Tullia Carmela Bruno, Aditi Kulkarni, , Riyue Bao

This study identifies tumor-intrinsic p38 activation as a driver of immune exclusion in epithelial cancers and suggests it as a therapeutic target to overcome immunotherapy resistance.

bioRxiv · Jun 16, 2026 · PreprintCancer Biology