Author

Qinchuan Wang

Johns Hopkins UniversityORCID 0000-0002-2359-3262Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

Elizabeth D. LuczakiD· 3 shared Gerald Warren HartiD· 2 shared Jonathan M. GrangeriD· 2 shared Liliana FloreaiD· 2 shared Mark E. AndersoniD· 2 shared Natasha Elizabeth ZacharaiD· 2 shared Neha Abrol· 2 shared Olurotimi O. MesubiiD· 2 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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3 shared papersMolecular Biology

Elizabeth D. LuczakORCID

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3 papers

Recent·Most cited

Chronic activation of a key exercise signal transducer, CaMKII, drives skeletal muscle aging and sarcopenia

Michael R. Bene, Tae Chung, Elizabeth D. Luczak, Gabriel A. Lopez-Cecetaite, William A. Fountain, Giovanni Rosales‐Soto, Erick O. Hernández‐Ochoa, Corina M. Antonescu, Liliana Florea, Seeun J. Jeong, Emily Elassal, Anne Le, Qian-Li Xue, Ahmet Höke, Peter M. Abadir, Qinchuan Wang

This study shows chronic CaMKII activation drives skeletal muscle aging and sarcopenia, and partial inhibition in aged muscle improves function and shifts the transcriptome toward a youthful state.

Aging Cell · Sep 1, 2026Peer-reviewedPhysiology

Excessive O-GlcNAcylation causes heart failure and sudden death

Priya Umapathi, Partha Sarathi Banerjee, Natasha Elizabeth Zachara, Neha Abrol, Qinchuan Wang, Olurotimi O. Mesubi, Elizabeth D. Luczak, Yuejin Wu, Jonathan M. Granger, An‐Chi Wei, Oscar E. Reyes Gaido, Liliana Florea, C. Conover Talbot, Gerald Warren Hart, Mark E. Anderson

This paper shows that excessive O-GlcNAcylation causes heart failure and sudden death in mice, while reducing it protects against cardiac stress.

Circulation · Feb 17, 2021Peer-reviewedMolecular Biology

O-GlcNAcylation, oxidation and CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes by distinct mechanisms

Olurotimi O. Mesubi, Adam G. Rokita, Neha Abrol, Yuejin Wu, Biyi Chen, Qinchuan Wang, Jonathan M. Granger, Anthony Tucker-Bartley, Elizabeth D. Luczak, Kevin R. Murphy, Priya Umapathi, Partha S. Banerjee, Tatiana N. Boronina, Robert Norman Cole, Lars Siegfried Maier, , Mark E. Anderson

This study shows that O-GlcNAcylation and oxidative stress via CaMKII contribute to atrial fibrillation in type 1 and type 2 diabetes through distinct mechanisms.

bioRxiv · Feb 18, 2020 · PreprintMolecular Biology