Author

Ming Jiang

Vanderbilt UniversityORCID 0000-0002-1927-5667Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Agnieszka E. GorskaiD· 1 shared Anand Kamal SinghiD· 1 shared Andrew J. MonteithiD· 1 shared Anna SkwarskaiD· 1 shared Benjamin J. ReismaniD· 1 shared Brian O. BachmanniD· 1 shared Eden G. TenBargeiD· 1 shared Haley Elizabeth RamseyiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Agnieszka E. GorskaORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintJul 13, 2026

Specific F1 ATP synthase inhibition delivers transient mitochondrial stress for selective targeting of acute myeloid leukemia

Matthew T. Villaume, Haley Elizabeth Ramsey, Valeria Impedovo…·Michael Robert Savona

This study shows that inhibiting F1 ATP synthase with EB2023 causes transient mitochondrial stress in AML cells, enabling effective treatment without harming healthy tissues.

DOI 10.64898/2026.07.10.737821

humanbulk RNA-seq

𝕏 PostBluesky