Author

Martina Bohuslavová

University of South Bohemia in České BudějoviceORCID 0000-0002-2015-7190Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:clustering· 1 Approach:statistical model· 1

Frequent Co-Authors

Alexander W. BruceiD· 3 shared Andrea HauserováiD· 3 shared Rebecca CollieriD· 3 shared Lenka GahurováiD· 2 shared Aleksandar I. MihajlovićiD· 1 shared Dávid DrutovičiD· 1 shared David PotěšiliD· 1 shared Joaquín Lilao‐GarzóniD· 1 shared

Activity by Year

2021-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersDevelopmental Biology

Alexander W. BruceORCID

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3 papers

Recent·Most cited

TEAD4 regulates apical domain homeostasis and cell-positioning to maintain the trophectoderm lineage during preimplantation mouse embryo development.

Rebecca Collier, Martina Bohuslavová, Michaela Vaškovičová, Aleksandar I. Mihajlović, Andrea Hauserová, Valeriya Zabelina, Monika Fluks, Lenka Gahurová, Dávid Drutovič, Alexander W. Bruce

This study shows TEAD4 maintains trophectoderm lineage integrity by regulating apical domain morphology and cell positioning in mouse preimplantation embryos.

bioRxiv · Nov 18, 2025 · PreprintDevelopmental Biology

Regulation of mouse blastocyst primitive endoderm differentiation by p38-mitogen-activated-kinases (p38-MAPKs) is inner-cell-mass (ICM) autonomous and unrelated to cavity expansion defects in ICM specification.

Martina Bohuslavová, Andrea Hauserová, Rebecca Collier, Joaquín Lilao‐Garzón, Silvia Muñoz Descalzo, Alexander W. Bruce

The paper investigates whether p38-MAPK inhibition affects primitive endoderm differentiation in mouse blastocysts through ICM autonomous mechanisms or cavity expansion defects.

bioRxiv · Sep 26, 2025 · PreprintDevelopmental Biology

DDX21 is a p38-MAPK sensitive nucleolar protein necessary for mouse preimplantation embryo development and cell-fate specification.

Pablo Bora, Lenka Gahurová, Andrea Hauserová, Martina Bohuslavová, Rebecca Collier, David Potěšil, Zbyněk Zdráhal, Alexander W. Bruce

This study investigates the role of DDX21 in mouse preimplantation embryo development, showing its necessity for cell-fate specification and blastocyst formation through p38-MAPK regulation.

Open Biology · Jul 1, 2021Peer-reviewedDevelopmental Biology