Author

Eva Jennische

University of GothenburgORCID 0000-0002-2147-8412Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

Dan Emil LindiD· 2 shared Jimmy Van den EyndeniD· 2 shared Marcus BorenäsiD· 2 shared Ruth Helen PalmeriD· 2 shared Adam Lehnberg· 1 shared Agata AniszewskaiD· 1 shared Arne ClaeysiD· 1 shared Beata Adamiak· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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2 shared papersCancer Biology

Dan Emil LindORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Glycoprotein G enables HSV-2 neuroinvasion and provides protection as a glycosylated vaccine antigen

Ebba Samuelsson, Carolina Gustafsson, Lindvi Gudmundsdotter, Ekaterina Mirgorodskaya, Jonas Nilsson, Maria Ekblad, Beata Adamiak, Eva Jennische, Stefan Lange, Edward Trybala, Staffan Görander, Tomas B. Bergström, Jan‐Åke Liljeqvist, Rickard Nordén

This study shows that HSV-2 glycoprotein G (mgG-2) is critical for neuroinvasion and that its glycosylation is essential for vaccine efficacy against HSV-2.

PLoS Pathogens · Jul 9, 2026Peer-reviewedMicrobiology

Spatial transcriptomics exploration of the primary neuroblastoma microenvironment unveils novel paracrine interactions

Joachim T. Siaw, Peter Merseburger, Marcus Borenäs, Caroline Jansson, Jenny Karlsson, Arne Claeys, Eva Jennische, Dan Emil Lind, David Gisselsson, Ruth Helen Palmer, Jimmy Van den Eynden

The paper uses spatial transcriptomics to analyze neuroblastoma microenvironments, identifying novel paracrine interactions between cancer cells and surrounding cells.

The Journal of Pathology · Aug 8, 2025Peer-reviewedCancer Biology

ALK/ATR combination therapy is effective in neuroblastoma mouse tumors driven by MYCN

Marcus Borenäs, Dan Emil Lind, Adam Lehnberg, Edit Zenténius, Matilda Esselius, Eva Jennische, Joel Johansson, Jikui Guan, Agata Aniszewska, Jimmy Van den Eynden, Bengt Hallberg, Ruth Helen Palmer

This study shows that combining ATR and ALK inhibitors improves survival in MYCN-amplified neuroblastoma mouse models lacking ALK mutations.

bioRxiv · Jul 17, 2025 · PreprintCancer Biology