Author

Gonzalo P. Solis

University of GenevaORCID 0000-0002-2359-3780Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Organism:Drosophila· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Vladimir L. KatanaeviD· 2 shared Camille Rabesahala de MeritensiD· 1 shared Mikhail KryuchkoviD· 1 shared Mikhail SavitskyiD· 1 shared Miriam StoeberiD· 1 shared Yonika Arum LarasatiiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

6 top collaborators · 9 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCell Biology

Vladimir L. KatanaevORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cell BiologyThe FASEB JournalPeer-reviewedDec 29, 2025

Visualizing the Dominant GPCR Coupling of Pathogenic Gαo Mutants in GNAO1-Related Disorders

Yonika Arum Larasati, Camille Rabesahala de Meritens, Miriam Stoeber…·Gonzalo P. Solis

This study uses a split-YFP BiFC assay to show that severe GNAO1 mutations impair GPCR disengagement, while milder mutants show near-normal receptor internalization.

4 citations

humanproteomics

𝕏 PostBluesky

GeneticsBiomedicinesPeer-reviewedOct 6, 2020

Humanization of Drosophila Gαo to model GNAO1 paediatric encephalopathies

Mikhail Savitsky, Gonzalo P. Solis, Mikhail Kryuchkov, Vladimir L. Katanaev

This paper humanizes the Drosophila Gαo gene to create a model for GNAO1-related paediatric encephalopathies, enabling functional studies of disease-causing mutations.

23 citations

Drosophilaexperimental assay

𝕏 PostBluesky