Author

Estefanía Moreno

Institut de Biomedicina de la Universitat de BarcelonaORCID 0000-0002-2491-5753Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:drug response / drug discovery· 2 Assay:electronic health records· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Vicent CasadóiD· 3 shared Enric I. CanelaiD· 2 shared Manuel GuzmániD· 2 shared Alba Hermoso-López· 1 shared Alexander F. HoffmaniD· 1 shared Alicia Álvaro-BlázqueziD· 1 shared Andrea Sánchez-RuiziD· 1 shared Andrés OzaitaiD· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersNeuroscience

Vicent CasadóORCID

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3 papers

Recent·Most cited

NeuroscienceEMBO Molecular MedicinePeer-reviewedMar 21, 2024

The CB1 receptor interacts with cereblon and drives cereblon deficiency-associated memory shortfalls

Carlos Costas‐Insua, Alba Hermoso-López, Estefanía Moreno…·Manuel Guzmán

This study identifies CB1R overactivation as a driver of CRBN deficiency-linked ARNSID and suggests CB1R blockade as a potential therapy.

3 citations

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintMay 31, 2021

A low repeated dose of Δ9-tetrahydrocannabinol affects memory performance through serotonergic signalling in mice

Lorena Galera-López, Victòria Salgado-Mendialdúa, Estefanía Moreno…·Andrés Ozaita

This study shows that repeated low-dose THC impairs memory in mice through serotonergic signaling via 5-HT2ARs, as evidenced by increased CB1R/5-HT2AR heterodimers and reduced LTP.

DOI 10.1101/2021.05.31.446448

mouseelectronic health records

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NeuroscienceeLifePeer-reviewedJun 8, 2020

Modulation of dopamine D1 receptors via histamine H3 receptors is a novel therapeutic target for Huntington’s disease

David Moreno‐Delgado, Mar Puigdellívol, Estefanía Moreno…·Peter James McCormick

This study identifies D1R-H3R heteromers as a therapeutic target for Huntington’s disease by showing their role in dopamine signaling and their modulation by H3R antagonists.

35 citations

𝕏 PostBluesky