Soni Lacefield

The paper compares the role of Stu2 and Ipl1 in error correction during meiosis and mitosis in budding yeast, finding that Stu2 is more critical for accurate chromosome segregation in meiosis.

The paper develops a mathematical model to show that Rim4 degradation and Ama1 translation are essential for irreversible meiotic exit in budding yeast.

The paper shows that in Mos-deficient eggs, polar bodies can re-enter the cell cycle and divide, leading to tumor formation due to disrupted MOS/MAPK signaling.

The paper identifies Clb4 as a factor preventing premature sister centromere separation in meiosis I of budding yeast, with effects dependent on genetic background.

This study shows that MOS signaling is essential for meiotic cell cycle regulation and maintaining the egg transcriptome required for developmental competence.

This study shows that cells lacking Bub3, a spindle checkpoint protein, retain specific chromosomes through aneuploidy, which provides a survival advantage by increasing the expression of genes involved in chromosome segregation.